Earl Dunbar wrote:
> Yes, but changing your diet to include a LOT more fats (the sausage
> worries me!), after having heart problems, would be risky in my mind.
>
> As for me, Lipitor seems to be working well, so I can enjoy Zweigels
> white hots, a Rochester signature "food". I balance everything with
> salmon and red wine.... more of the latter.
-----------------------------------------------------------
<http://www.amazon.com/exec/obidos/tg/detail/-/0967089700/qid=1125063510/sr=8-1/ref=pd_bbs_1/104-9279265-7494304?v=glance&s=books&n=507846>
Read this and then stop worrying about the sausage or, more
specifically, about your cholesterol. As I said, the cholesterol
emperor has no clothes. If you don't want to invest in the book take a
look at the first few reviews of the book. They'll tell you in a
nutshell what it's all about.
If you're over age 50 ask your doctor to provide you with the title and
author of *any* scientific study that shows that high cholesterol is a
signigicant risk factor for CHD for men over the age of 50. Make sure
the study contains the actual data since many studies reference other
studies which do not actually support the assertions made. So much for
peer reviewed medical science. Tracking the truth and source data in
some of these papers is like trying to locate a spammer.
As to Lipitor, I hope it all works out for you. For myself, I've taken
it for 7 years and finally decided that it is probably doing me more
harm than good. I didn't even get to the part about coenzyme Q10 yet.
<http://faculty.washington.edu/~ely/coenzq10.html> coenzyme Q10 is a
vitamin present in foods and naturally produced by the body. It is
critical to proper cellular (mitochondrial) function of all living
things. It so happens that the chemical pathways in your body which are
responsible for cholesterol production are also responsible for
coenzympe Q10 production. When you take statin drugs your coenzyme Q10
production is cut right along with your cholesterol.
A quote from the paper above. Be sure to read the third paragraph
--------------------------------------------------------------------
COENZYME Q10 DEFICIENCY
Normal blood and tissue levels of CoQ10 have been well established by
numerous investigators around the world. Significantly decreased levels
of CoQ10 have been noted in a wide variety of diseases in both animal
and human studies. CoQ10 deficiency may be caused by insufficient
dietary CoQ10, impairment in CoQ10 biosynthesis, excessive utilization
of CoQ10 by the body, or any combination of the three. Decreased dietary
intake is presumed in chronic malnutrition and cachexia(12).
The relative contribution of CoQ10 biosynthesis versus dietary CoQ10 is
under investigation. Karl Folkers takes the position that the dominant
source of CoQ10 in man is biosynthesis. This complex, 17 step process,
requiring at least seven vitamins (vitamin B2 - riboflavin, vitamin B3 -
niacinamide, vitamin B6, folic acid, vitamin B12, vitamin C, and
pantothenic acid) and several trace elements, is, by its nature, highly
vulnerable. Karl Folkers argues that suboptimal nutrient intake in man
is almost universal and that there is subsequent secondary impairment in
CoQ10 biosynthesis. This would mean that average or "normal" levels of
CoQ10 are really suboptimal and the very low levels observed in advanced
disease states represent only the tip of a deficiency "ice berg".
HMG-CoA reductase inhibitors used to treat elevated blood cholesterol
levels by blocking cholesterol biosynthesis also block CoQ10
biosynthesis(13). The resulting lowering of blood CoQ10 level is due to
the partially shared biosynthetic pathway of CoQ10 and cholesterol. In
patients with heart failure this is more than a laboratory observation.
It has a significant harmful effect which can be negated by oral CoQ10
supplementation(14).
----------------------------------------------------------------------
Good luck.
Chuck Norcutt
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